Corrigendum: Mutations in CDCA7 and HELLS cause immunodeficiency–centromeric instability–facial anomalies syndrome

نویسندگان

  • Peter E. Thijssen
  • Yuya Ito
  • Giacomo Grillo
  • Jun Wang
  • Guillaume Velasco
  • Hirohisa Nitta
  • Motoko Unoki
  • Minako Yoshihara
  • Mikita Suyama
  • Yu Sun
  • Richard J. L. F. Lemmers
  • Jessica C. de Greef
  • Andrew Gennery
  • Paolo Picco
  • Barbara Kloeckener-Gruissem
  • Tayfun Güngör
  • Ismail Reisli
  • Capucine Picard
  • Kamila Kebaili
  • Bertrand Roquelaure
  • Tsuyako Iwai
  • Ikuko Kondo
  • Takeo Kubota
  • Monique M. van Ostaijen-Ten Dam
  • Maarten J. D. van Tol
  • Corry Weemaes
  • Claire Francastel
  • Silvère M. van der Maarel
  • Hiroyuki Sasaki
چکیده

The life-threatening Immunodeficiency, Centromeric Instability and Facial Anomalies (ICF) syndrome is a genetically heterogeneous autosomal recessive disorder. Twenty percent of patients cannot be explained by mutations in the known ICF genes DNA methyltransferase 3B or zinc-finger and BTB domain containing 24. Here we report mutations in the cell division cycle associated 7 and the helicase, lymphoid-specific genes in 10 unexplained ICF cases. Our data highlight the genetic heterogeneity of ICF syndrome; however, they provide evidence that all genes act in common or converging pathways leading to the ICF phenotype.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Mutations in CDCA7 and HELLS cause immunodeficiency–centromeric instability–facial anomalies syndrome

Supplementary Figure 1: Repeat hypomethylation in ICF patients from families E, F and G (a, b) Sat-α methylation analysis using HhaI digestion followed by Southern blot analysis of healthy controls and patients 2.1 from family E and 2.2 from family F showed hypomethylation in the patient derived samples. (c) SatII (BstBI) and Sat-α (HhaI) methylation analysis by Southern blot analysis in DNA de...

متن کامل

Combined immunodeficiency develops with age in Immunodeficiency-centromeric instability-facial anomalies syndrome 2 (ICF2)

The autosomal recessive immunodeficiency-centromeric instability-facial anomalies syndrome (ICF) is characterized by immunodeficiency, developmental delay, and facial anomalies. ICF2, caused by biallelic ZBTB24 gene mutations, is acknowledged primarily as an isolated B-cell defect. Here, we extend the phenotype spectrum by describing, in particular, for the first time the development of a combi...

متن کامل

Immunodeficiency, centromeric heterochromatin instability of chromosomes 1, 9, and 16, and facial anomalies: the ICF syndrome.

Instability of the heterochromatic centromeric regions of chromosomes 1, 9, and 16 associated with immunodeficiency was found in a four year old girl. Similar phenotypic and chromosomal abnormalities were described in a previous patient studied by us and in four other published cases. All these patients have facial anomalies in addition to combined immunodeficiency and chromosomal instability. ...

متن کامل

Immunodeficiency, centromeric region instability, facial anomalies syndrome (ICF)

The Immunodeficiency, Centromeric region instability, Facial anomalies syndrome (ICF) is a rare autosomal recessive disease described in about 50 patients worldwide and characterized by immunodeficiency, although B cells are present, and by characteristic rearrangements in the vicinity of the centromeres (the juxtacentromeric heterochromatin) of chromosomes 1 and 16 and sometimes 9. Other varia...

متن کامل

A case with ICF syndrome lost to rubella pneumonitis.

The immunodeficiency, centromeric instability, and facial anomalies (ICF) syndrome is a rare autosomal recessive disorder characterized by variable immunodeficiency, instability of the pericentromeric heterochromatin, and facial dysmorphism. Here we report a new case of ICF syndrome who died of rubella pneumonitis. A six year-old-girl who was the first child of consanguineous parents was admitt...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره 7  شماره 

صفحات  -

تاریخ انتشار 2015